- 面议
起订量：
人脐静脉血管内皮细胞HUVEC
- 型号
该企业相似产品
本公司 是一家专注于生命科学和生物技术领域的高科技企业，专业提供分子生物学、免疫学、生命科学基础研究以及临床检测等诸多领域的试剂、耗材、仪器等各类产品及生物技术服务。
公司一方面注意跟踪世界范围分子生物学、细胞生物学等生物学科的发展前沿，努力将欧美专业生产厂家的具有*水平的产品*给各类研究人员；另一方面也非常重视自主产品研究和开发，推出了一系列具有竞争力的产品和服务。公司客户遍布大学、研究所、医院、卫生防疫、商品检验检疫、制药公司、生物技术公司和食品工业等单位。
公司在重视产品质量的同时，也建立了一套集技术支持、物流、售后服务等多个部门的*的服务体系，努力把我们方便、快捷、周到的服务提供给每一个客户。
主要代理产品：
试剂盒：免疫组化试剂盒、ELISA试剂盒、分子生物学试剂盒。
细胞：*细胞、国产肿瘤细胞、国产正常细胞。
耗材：细胞培养耗材、普通实验耗材。
生化试剂：*生化试剂、进口分装生化试剂、国产生化试剂。
抗体：免疫组化、免疫荧光、流式细胞检测、免疫细胞化学。
公司主推品牌：
Amresco 、Sigma、 Peprotech 、Corning 、BD Faclon 、Axygen、 Invitrogen、 Qiagen、 Hyclone 、Uscnlife、 Wako 、SantaCruz、sciencell、Gibco、R&D、Linco、Amersham、Millipore、Costar
详细信息
*培养基:（FM, Cat. No. 2301）
储存：液氮
运输：干冰
用途：科研
The most abundant cell type in lung interstitium is fibroblasts. They resemble ordinary fibroblasts but have some distinguishing features, for example, they have long branching processes and gap junctions. Their principle function is production of type III collagen, elastin, and proteoglycans of the extracellular matrix of the alveolar septa. Pulmonary fibroblasts （PF） play an important role in the repair and remodeling processes following injury. The controlled accumulation of fibroblasts to sites of inflammation is crucial to effective tissue repair after injury [1]. Either inadequate or excessive accumulation of fibroblasts could result in abnormal tissue function. For example, the excess proliferation of fibroblasts contributes to the adventitial thickening observed during the development of hypoxia-induced pulmonary hypertension [2].
HPF from ScienCell Research Laboratories are isolated from human lung tissue. HPF are cryopreserved at primary culture and delivered frozen. Each vial contains >5 x 105 cells in 1 ml volume. HPF are characterized by immunofluorescent method with antibody to fibronectin. HPF are negative for HIV-1, HBV, HCV, mycoplasma, bacteria, yeast and fungi. HPF are guaranteed to further expand for 15 population doublings at the conditions provided by ScienCell Research Laboratories.
Reference
[1] Kuwano K, Hagimoto N, Hara N. （2001） Molecular mechanisms of pulmonary fibrosis and current treatment. Curr Mol Med 1（5）:551-73.
[2] Das M, Dempsey EC, Reeves JT, Stenmark KR. （2004） Selective expansion of fibroblast subpopulations from pulmonary artery adventitia in response to hypoxia. Am J Physiol Lung Cell Mol Physiol 282（5）:L976-86
