AAT抗胰蛋白酶(兔多克隆抗体)

AAT抗胰蛋白酶(兔多克隆抗体)

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2017-12-11 15:24:00
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广州欧边生物制品有限公司

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AAT抗胰蛋白酶(兔多克隆抗体)α-1-Antitrypsin 我司为大家提供各种生物原料免疫组化产品,欢迎大家随时咨询。

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AAT抗胰蛋白酶(兔多克隆抗体)

α-1-Antitrypsin 

广州健仑生物科技有限公司

AAT是一种存在于正常人血清中的糖蛋白,可以标记组织细胞与网状细胞。AAT的阳性染色可用来检测恶性纤维组织细胞瘤,也可用于筛查隐源性肝硬化和其它不明原因的门静脉纤维化的肝脏疾病。免疫组化学检测AAT还可用于检测遗传性AAT缺陷,良、恶性肝脏肿瘤,卵黄癌等。

我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

欢迎咨询

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【产品介绍】

细胞定位:细胞浆

适用组织:石蜡/冰冻

阳性对照:扁桃体

抗原修复:热修复(EDTA)

抗体孵育时间:30-60min

产品编号产品名称克隆型别
OB001AACT(抗胰糜蛋白酶)polyclonal
OB002AAT(抗胰蛋白酶)polyclonal
OB003ACTH(促肾上腺皮质激素)polyclonal
OB004Actin,Muscle Specific(肌肉特异性肌动蛋白)HHF35
OB005Actin,Smooth Muscle(平滑肌肌动蛋白)1A4
OB006AFP(甲胎蛋白)polyclonal

 

AAT抗胰蛋白酶(兔多克隆抗体)

阿米巴类感染是由根足虫纲(Rhizopoda)、阿米巴目(Amoebina)、内阿米巴科(Ent—amoebidae)、内阿米巴属(Entamoeba)下各种内阿米巴所引起的,临床习惯简称阿米巴。其中肠道阿米巴原虫,种类虽多,大多寄生于人体内作为共居生物而无致病能力,唯有溶组织内阿米巴寄生于人体后,在一定条件下,可引起疾病,被认为是有致病力的阿米巴。
病原学
溶组织内阿米巴有滋养体及包囊两期。滋养体自包囊逸出后寄生于大肠肠腔或肠壁,以大肠内容物包括细菌为养料,借肠内乏氧和存在细菌的条件,进行分裂繁殖。滋养体大小不一,12~60um,而以15~30um为常见。
滋养体抵抗力甚弱,在室温下数小时内死亡,遇稀盐酸则在数分钟内死亡。滋养体在适当条件下能侵袭与破坏组织,造成结肠病变,引起临床症状,所以滋养体是溶组织内阿米巴的侵袭型,但它无感染能力。因为在体外它很快死亡,即使进入消化道也很快被胃酸破环。包囊抵抗外界能力很强,在大便中能存活2周以上,在水中能存活5周,能耐受常用化学消毒剂的作用。但对热和干燥较敏感,加热至50℃几分钟即死。包囊可随粪便排到外界。人于吞食被包囊污染的食物或水即造成感染。所以溶组织内阿米巴的感染型是包囊。包囊被吞食后,不受胃酸破环,经胃达回肠。由于小肠碱性消化液的作用及虫体的活动,含有四核的虫体从囊壁逸出。虫体又经一系列的复杂变化后,分裂为四个至八个小滋养体,定居于盲肠和大肠近端。
发病原因
包囊被吞食后进入小肠下段,滋养体脱囊逸出,随粪便下降,寄居于盲肠、结肠、直肠等部营共居生活,以肠腔内细菌及浅表上皮细胞为食。在适宜的条件下,滋养体侵袭肠粘膜,造成溃疡,到一定范围和程度时,酿成痢疾。
流行病学
本病见于*各地,其感染率的高低是同各地环境卫生和居民营养状况等关系极大。溶组织内阿米巴病在热带、亚热带、温带地区,发病较多,以秋季为多,夏季次之。发病率农村高于城市,男子多于女子,成年多于儿童,幼儿患者很少,可能与吞食含包囊食物机会的多少有关。

我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

想了解更多的产品及服务请扫描下方二维码:

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

【】 
【腾讯  】 
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

The amoebiasis is caused by a variety of endoenterprises such as Rhizopoda, Amoebina, Ent-amoebidae and Entamoeba Cause, clinical practice referred to as amoeba. Including intestinal amoeba protozoa, although many species, most of the parasites in the human body as living organisms without pathogenic capacity, the only solution Entamoeba histolytica parasitic on the human body, under certain conditions, can cause disease, is considered Is a virulent amoeba.
Etiology
Entamoeba histolytica trophozoites and cysts two. Trophoblast escaping from the cyst after the parasitic intestinal or intestinal wall of the large intestine, including the contents of the large intestine, including bacteria as nutrients, by intestinal hypoxia and the presence of bacteria in the conditions of division and reproduction. Trophoblast sizes, 12 ~ 60um, and 15 ~ 30um as common.
Trophozoites resistance is very weak, died within a few hours at room temperature, the case of dilute hydrochloric acid died within a few minutes. Trophoblast invasion and destruction under appropriate conditions in the organization, causing colonic lesions, causing clinical symptoms, so trophozoites Entamoeba histolytica invasion, but it has no ability to infection. Because it quickly dies in vitro, it quickly breaks down even when it enters the digestive tract. Encapsulated resistance to the outside world is very strong, can survive in the stool for more than 2 weeks, can survive in water for 5 weeks, can tolerate the role of common chemical disinfectants. But more sensitive to heat and dry, heated to 50 ℃ a few minutes to die. Encapsulation can be excreted with the outside world. People who ingest swallowed contaminated food or water cause infection. So Entamoeba histolytica infection is cysts. Cysts are swallowed, not by the acid broken ring, up to the stomach by the ileum. As a result of the role of small intestinal alkaline digestive juice and the activity of parasites, tetrazoite-containing parasites escape from the capsule wall. After a series of complex changes in parasites, split into four to eight small trophozoites, settled in the cecum and the proximal colon.
Causes
Encapsulated into the lower intestine after being swallowed, nourish the body to escape, with the drop in feces, colonization in the cecum, colon, rectum and other camps living together to intestinal bacteria and superficial epithelial cells for food. Under suitable conditions, trophozoites attack the intestinal mucosa, causing ulcers, to a certain extent and extent, resulting in diarrhea.
Epidemiology
The disease seen in all parts of the world, the level of infection is highly related to the health status of all localities and residents with great nutritional status. Tissue Entamoeba histolytica in tropical, subtropical, temperate regions, more incidence, with more fall, summer followed. Morbidity is higher in rural areas than in urban areas, with more males than females, more adulthood than children, and very few toddlers, which may be related to the chance of swallowed cysts containing food.

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